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Horse Disorders and Diseases
Digestive Disorders of Horses
Gastrointestinal Obstruction (Blockages) in Horses
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Chapters in Horse Disorders and Diseases
  • Blood Disorders of Horses
  • Heart and Blood Vessel Disorders of Horses
  • Digestive Disorders of Horses
  • Hormonal Disorders of Horses
  • Eye Disorders of Horses
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  • Bone, Joint, and Muscle Disorders in Horses
  • Brain, Spinal Cord, and Nerve Disorders of Horses
  • Reproductive Disorders of Horses
  • Lung and Airway Disorders of Horses
  • Skin Disorders of Horses
  • Kidney and Urinary Tract Disorders of Horses
  • Metabolic Disorders of Horses
  • Disorders Affecting Multiple Body Systems of Horses
Topics in Digestive Disorders of Horses
  • Introduction to Digestive Disorders of Horses
  • Congenital and Inherited Disorders of the Digestive System in Horses
  • Dental Development of Horses
  • Dental Disorders of Horses
  • Disorders of the Mouth in Horses
  • Pharyngeal Paralysis in Horses
  • Disorders of the Esophagus in Horses
  • Stomach (Gastric) Ulcers in Horses
  • Gastrointestinal Obstruction (Blockages) in Horses
  • Colic in Horses
  • Intestinal Disorders Other than Colic in Horses
  • Gastrointestinal Parasites of Horses
  • Disorders of the Liver in Horses
  • Disorders of the Rectum and Anus in Horses
 
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Gastrointestinal Obstruction (Blockages) in Horses

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Intestinal obstructions (blockages) are common in horses. Blockages can occur in any part of the digestive tract and can cause sharp and severe pain. These obstructions can stop the flow of food through the digestive tract and, in some instances, impair blood flow or cause damage to intestinal tissues.

It is not always possible to determine the cause of an individual blockage. Feed impactions, parasite infections or migrations, abnormal dental conditions, and changes in diet, daily activities, medications, or sudden stress may play a role in functional obstructions. Coarse feeds, reduced water consumption, and eating foreign objects are other common factors. Blockage may occur in a variety of locations within the intestines.

Twists, muscle tension changes, and changes in the position of various parts of the intestines can also bring on impaction or blockage. Altered intestinal movement and, possibly, strenuous exercise and rolling may cause or contribute to obstructions. Broodmares may be predisposed to obstructions, especially during pregnancy and shortly after giving birth. Standardbred stallions and colts develop inguinal and scrotal hernias more commonly than other breeds.

Pain is the most common sign of intestinal obstruction in horses. The horse may pace, stretch, kick at its abdomen, and, upon occasion, roll or vocalize. Otherwise, the signs are the same as for colic (see Digestive Disorders of Horses: Colic in Horses).

For functional obstructions in horses, the treatment is the same as for colic (see Digestive Disorders of Horses: Colic in Horses), and may include medicine to relieve pain, fluid treatment, and intestinal lubricants and possibly laxatives. Mechanical obstructions such as a twisted intestine frequently require surgery to make a definitive diagnosis. If the obstruction is a foreign (nonfood) object that has penetrated the digestive system, care needs to be taken to locate and repair the injuries (there is often more than one injured site). This normally requires surgery. Antibiotics are usually prescribed before surgery to reduce infection. Additional supportive treatment may include fluids, electrolytes (salts), and calcium. Horses that require exploratory abdominal surgery to locate and correct an obstruction have an overall longterm survival rate of 50%, but early surgical intervention can improve that outlook.

Prevention of all—or even most—cases of intestinal obstruction is not possible. However, the likelihood is reduced if changes in diet and daily routines are gradual, adequate water is available at all times, dental care is regular and appropriate, and parasites are controlled. Access to coarse feeds and foreign materials should be avoided or corrected.

Last full review/revision July 2011 by Peter D. Constable, BVSc (Hons), MS, PhD, DACVIM; Gordon J. Baker, BVSc, PhD, MRCVS, DACVS; Joseph A. DiPietro, DVM, MS; Walter Ingwersen, DVM, DVSc, DACVIM; John E. Madigan, DVM, MS; James N. Moore, DVM, PhD; Michael J. Murray, DVM, MS; Sofie Muylle, DVM, PhD; Stanley I. Rubin, DVM, MS, DACVIM; Susan D. Semrad, VMD, PhD, DACVIM; Josie L. Traub-Dargatz, DVM, MS, DACVIM

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