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Horse Disorders and Diseases
Eye Disorders of Horses
Disorders of the Cornea in Horses
Corneal Ulcers
Corneal Abscesses
Corneal Lacerations
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  • Dog Disorders and Diseases
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Chapters in Horse Disorders and Diseases
  • Blood Disorders of Horses
  • Heart and Blood Vessel Disorders of Horses
  • Digestive Disorders of Horses
  • Hormonal Disorders of Horses
  • Eye Disorders of Horses
  • Ear Disorders of Horses
  • Immune Disorders of Horses
  • Bone, Joint, and Muscle Disorders in Horses
  • Brain, Spinal Cord, and Nerve Disorders of Horses
  • Reproductive Disorders of Horses
  • Lung and Airway Disorders of Horses
  • Skin Disorders of Horses
  • Kidney and Urinary Tract Disorders of Horses
  • Metabolic Disorders of Horses
  • Disorders Affecting Multiple Body Systems of Horses
Topics in Eye Disorders of Horses
  • Eye Structure and Function in Horses
  • Disorders of the Eyelids in Horses
  • Disorders of the Nasal Cavity and Tear Ducts in Horses
  • Disorders of the Conjunctiva in Horses
  • Disorders of the Cornea in Horses
  • Disorders of the Anterior Uvea in Horses
  • Glaucoma in Horses
  • Disorders of the Lens in Horses
  • Disorders of the Retina, Choroid, and Optic Disk (Ocular Fundus) in Horses
  • Disorders of the Optic Nerve in Horses
  • Prolapse of the Eye in Horses
  • Eyeworm Disease (Thelaziasis) in Horses
  • Cancers and Tumors of the Eye in Horses
 
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Disorders of the Cornea in Horses

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The cornea protects the front of the eye and is also important in focusing light on the retina at the back of the eye. Because the cornea is critical for proper vision, it is important to address any disorders or injuries promptly.

Corneal Ulcers

Corneal ulcers are common in horses. This disorder has the potential to affect vision unless the cause is promptly diagnosed and treated. Many equine corneal ulcers occur as a result of injury to the eye, with inflammation of the cornea (called keratitis) that ranges from superficial to deep. Superficial ulcers are usually controlled with topical antibiotics and correction of any mechanical factors. In addition, veterinarians often prescribe medications to reduce eye pain.

Corneal ulcers may be complicated by a fungal invasion; this is termed equine ulcerative keratomycosis. The fungus, which is normally present in the conjunctiva, multiplies rapidly after injury to the cornea and causes inflammation and ulcers. The diagnosis is confirmed by identifying the fungus in cells from the cornea. Treatment must begin promptly to avoid vision loss and includes both therapy with antifungal drugs and surgery. Even with aggressive treatment, vision after keratomycosis is lost in about 25% of affected eyes.

Syndromes of very slow-healing and recurrent superficial ulcers also occur in horses. In such cases, a herpesvirus is often the cause. Initial treatment involves removal of the dead, damaged, or infected tissue of the ulcer, followed by prescription topical medication.

Corneal Abscesses

Pus-filled sores in the connective tissue of the cornea (corneal stromal abscesses) in horses may be caused by healing ulcers or defects of the cornea and the trapping of bacteria or fungi (or both) within the connective tissue after healing tissue is formed. A white to yellow material in the connective tissue is surrounded by an intense inflammation and swelling of the cornea and formation of blood vessels. In addition, there may be a variable but sometimes intense inflammation of the anterior uvea. Treatments include topical and, in some cases, whole-body antibiotics, antifungal drugs, drugs to reduce pain, and nonsteroidal anti-inflammatory drugs. In addition, surgery may be required.

Corneal Lacerations

Minor lacerations of the cornea are common in horses and can usually be treated with topical antibiotics and other drugs as recommended by your veterinarian. Severe lacerations or perforations of the cornea often require surgery and more aggressive therapy. Signs of laceration include swelling or prolapse of the iris, swelling of the ciliary bodies, and blood in the eye.

Last full review/revision July 2011 by Kirk N. Gelatt, VMD; David G. Baker, DVM, MS, PhD, DACLAM; Steven R. Hollingsworth, DVM, DACVO

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