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In This Topic
Ear, Nose, and Throat Disorders
Middle Ear and Tympanic Membrane Disorders
Traumatic Perforation of the Tympanic Membrane
Symptoms and Signs
Diagnosis
Treatment
Key Points
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Chapters in Ear, Nose, and Throat Disorders
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  • Hearing Loss
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Topics in Middle Ear and Tympanic Membrane Disorders
  • Introduction
  • Mastoiditis
  • Myringitis
  • Otitis Media (Acute)
  • Otitis Media (Secretory)
  • Otitis Media (Chronic)
  • Otic Barotrauma
  • Otosclerosis
  • Traumatic Perforation of the Tympanic Membrane
     
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    Traumatic Perforation of the Tympanic Membrane

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    Traumatic perforation of the tympanic membrane (TM) can cause pain, bleeding, hearing loss, tinnitus, and vertigo. Diagnosis is based on otoscopy. Treatment often is unnecessary. Antibiotics may be needed for infection. Surgery may be needed for perforations persisting > 2 mo, disruption of the ossicular chain, or injuries affecting the inner ear.

    Traumatic causes of TM perforation include

    • Insertion of objects into the ear canal purposely (eg, cotton swabs) or accidentally
    • Concussion caused by an explosion or open-handed slap across the ear
    • Head trauma (with or without basilar fracture)
    • Sudden negative pressure (eg, strong suction applied to the ear canal)
    • Barotrauma (eg, during air travel or scuba diving)
    • Iatrogenic perforation during irrigation or foreign body removal

    Penetrating injuries of the TM may result in dislocations of the ossicular chain, fracture of the stapedial footplate, displacement of fragments of the ossicles, bleeding, a perilymph fistula from the oval or round window resulting in leakage of perilymph into the middle ear space, or facial nerve injury.

    Symptoms and Signs

    Traumatic perforation of the TM causes sudden severe pain sometimes followed by bleeding from the ear, hearing loss, and tinnitus. Hearing loss is more severe if the ossicular chain is disrupted or the inner ear is injured. Vertigo suggests injury to the inner ear. Purulent otorrhea may begin in 24 to 48 h, particularly if water enters the middle ear.

    Diagnosis

    • Otoscopy
    • Audiometry

    Perforation is generally evident on otoscopy. Any blood obscuring the ear canal is carefully suctioned. Irrigation and pneumatic otoscopy are avoided. Extremely small perforations may require otomicroscopy or middle ear impedance studies for definitive diagnosis. If possible, audiometric studies are done before and after treatment to avoid confusion between trauma-induced and treatment-induced hearing loss.

    Photographs

    Traumatic Perforation of Tympanic Membrane

    Traumatic Perforation of Tympanic Membrane

    Patients with marked hearing loss or severe vertigo are evaluated by an otolaryngologist as soon as possible. Exploratory tympanotomy may be needed to assess and repair damage. Patients with a large TM defect should also be evaluated, because the displaced flaps may need to be repositioned.

    Treatment

    • Ear kept dry
    • Oral or topical antibiotics if dirty injury

    Often, no specific treatment is needed. The ear should be kept dry; routine antibiotic ear drops are unnecessary. However, prophylaxis with oral broad-spectrum antibiotics or antibiotic ear drops is necessary if contaminants may have entered through the perforation as occurs in dirty injuries.

    If the ear becomes infected, amoxicillinSome Trade Names
    AMOXIL
    TRIMOX
    Click for Drug Monograph
    500 mg po q 8 h is given for 7 days.

    Although most perforations close spontaneously, surgery is indicated for a perforation persisting > 2 mo. Persistent conductive hearing loss suggests disruption of the ossicular chain, necessitating surgical exploration and repair.

    Key Points

    • Many perforations are small and heal spontaneously.
    • The ear should be kept dry during healing; topical or systemic antibiotics are unnecessary unless there is significant contamination or if infection develops.
    • Surgery is done to repair damage to the ossicles and for perforations persisting > 2 mo.

    Last full review/revision December 2012 by Richard T. Miyamoto, MD

    Content last modified January 2013

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